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Metoclopramida gotas dosis en adultos ? [on estudio, 2008 (Espanões 2)] by Dr. Eduardo Hernández, Prof. Carlos Antonio Salinas-Cabral (University Hospital of La Laguna, Rizal, Philippines). Abstract: Adult rats are exposed for 12 weeks to the following: a single intraperitoneal injection of subcutaneous dose 50 mg/lb (i.p.) of i.p. clopramide; an intrathecal injection of 10 µg (0,5 mg/kg) difenoxin (4 × 10(-5) M) administered 1 h before the subcutaneous injection of i.p. clopramide; and an oral dose of 0.2 mg/kg (3×10(-6)/kg) l-tyrosine or diphosphonate. The treatment group received diphospho-dose twice a month. treatment-naïve control group was given i.p. saline placebo and the two doses of clopramide, and the two groups the generic pharmacy contact number of control were given i.p. saline placebo and each of four treatment regimens. Tolerance was observed to clopramide and l-tyrosine within 2 weeks in the treatment group after dosing at 50 mg/lb and 8 mg/kg (0,5 mg/kg, i.p.) of 1 h before the l-tyrosine regimen. When treatment was administered at 0.2 mg/kg (3×10(-6)/kg) of l-tyrosine, no further tolerance was observed to clopramide and l-tyrosine in the treatment group, indicating presence of a dose-dependent tolerance. The drug tolerance effect was not related to the effect of clopramide on dopamine system, nor was it related to the effect of clopramide on serotonin system. Tolerance to clopramide, which was dose dependent, persisted after two treatments, i.p. and intramuscularly, of the diphospholipid. Tolerance to clopramide was not dose dependent. This suggests that the tolerance of clopramide to is not caused by its effects on a number of neurotransmitters except its effects on the dopamine system. http://www.ncbi.nlm.nih.gov/pubmed/19724883 Dopa for depression and anxiety in patients with Parkinson's disease: a randomized, double-blind, placebo-controlled trial. By: R.B. Pardridge. August 2007. (N-Vital; Aug 07) http://www.ncbi.nlm.nih.gov/pubmed/18512170 Clonidine for the Treatment of Neuropathic Symptoms Parkinson's Disease: A Double-Blind, Placebo-Controlled Clinical Study By: K.J. Sauer and E.T. Schmader. January 2007. (Neurology; Jan 7, 2007) http://www.ncbi.nlm.nih.gov/pubmed/18408051 Statin in Parkinson's Disease http://www.hospitalsociety.org/publications/Parkinson-Drug/Parkinson-Drug.pdf Fluoxetine in the treatment of Parkinson's Disease A double-blind, active-placebo, parallel-group, placebo-controlled study was undertaken. Twenty patients were treated with either fluoxetine (n = 18) or placebo 18), and their symptoms scores of were recorded at baseline and 4 weeks after completion of 6 months treatment. At each time point, the participants took medication they had been taking for the previous 24 weeks (approximately twice a day). A decrease in the mean scores on Parkinsonian Rating Scale (Patient Health Questionnaire – Revised) from baseline occurred with fluoxetine. In contrast, the improvement seen fluoxetine-treated patients remained evident and statistically significant during the entire study-term. An increase in the number of "improved" symptoms was obtained from 4 weeks to 6 months in patients on fluoxetine. The investigators considered that promethazine codeine canada otc reduction of symptoms obtained in fluoxetine-treated patients could have been in large measure due to placebo effects. They concluded that additional clinical studies are needed to establish whether placebo-controlled fluoxetine studies demonstrate the possibility of reducing symptom severity in Parkinson's disease. http://onlinelibrary.wiley.com/doi/10.1002/hcp.25116/abstract Effect of a Selective Dopaminergic Serotonin Reuptake Inhibitor for Treatment of Depression

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